Please use this identifier to cite or link to this item: http://repo.knmu.edu.ua/handle/123456789/32910
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dc.contributor.authorTychonova, Liudmila-
dc.contributor.authorTernopol, Yuliia-
dc.date.accessioned2023-11-16T15:02:49Z-
dc.date.available2023-11-16T15:02:49Z-
dc.date.issued2023-03-16-
dc.identifier.citationTychonova L. V. Neurologic complications due to vitamin deficiencies after bariatric surgery/ L. V. Tychonova, Yu. O. Ternopol // Scientific Collection «InterConf». ─ 2023. ─ № 146: Proceedings of the 2nd International Scientific and Practical Conference «Progressive Science and Achievements» (March 16─18, 2023), Doha, Qatar. ─ P. 289─294.en_US
dc.identifier.isbn978-9927-156-34-2-
dc.identifier.urihttp://repo.knmu.edu.ua/handle/123456789/32910-
dc.description.abstractBariatric surgery is the most effective method of treating morbid obesity, but after its performance the patient may develop vitamin deficiency. Against the background of insufficient food intake and absorption disorders after the surgery, the deficiency of vitamins B1, B6, B12 and vitamin E is observed first of all, which leads to complications in the form of central and peripheral nervous system dysfunctions. The most pronounced changes of the nervous system are observed with vitamin B1 deficiency in the form of polyneuropathy and severe Wernicke's encephalopathy. Vitamin B12 deficiency is clinically manifested by a picture of funicular myelosis and polyneuropathy. Insufficient intake of vitamin B6 and vitamin E leads to polyneuropathy. Only the early start of treatment guarantees a favorable outcome of the neurological deficit.en_US
dc.language.isoenen_US
dc.subjectobesityen_US
dc.subjectbariatric surgeryen_US
dc.subjectvitamin deficiencyen_US
dc.subjectWernicke's encephalopathyen_US
dc.subjectpolyneuropathyen_US
dc.subject2023aen_US
dc.titleNeurologic complications due to vitamin deficiencies after bariatric surgeryen_US
dc.typeThesisen_US
Appears in Collections:Наукові праці. Кафедра неврології

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