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\par }\pard \qc \li0\ri0\sl360\slmult1\widctlpar\aspalpha\aspnum\faauto\adjustright\rin0\lin0\itap0\pararsid10436759 {\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid10436759 \hich\af0\dbch\af37\loch\f0 Delevskaya V.Yu.
\par }\pard \qj \li0\ri0\sl360\slmult1\widctlpar\aspalpha\aspnum\faauto\adjustright\rin0\lin0\itap0\pararsid10436759 {\b\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid10436759 \hich\af0\dbch\af37\loch\f0 To 
\hich\af0\dbch\af37\loch\f0 the \hich\af0\dbch\af37\loch\f0 f\hich\af0\dbch\af37\loch\f0 eatures of intrac\hich\af0\dbch\af37\loch\f0 ardiac hemodynamics in patients with arterial hypertension and chronic obstructive pulmonary disease
\hich\af0\dbch\af37\loch\f0 .
\par }\pard \qj \fi708\li0\ri0\sl360\slmult1\widctlpar\aspalpha\aspnum\faauto\adjustright\rin0\lin0\itap0\pararsid7802150 {\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid10436759\charrsid10436759 
\par }{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid10436759 \hich\af0\dbch\af37\loch\f0 Khar\hich\af0\dbch\af37\loch\f0 kiv National Medical University, \hich\af0\dbch\af37\loch\f0 \hich\f0 Department of Internal Medicine \'b9
\loch\f0  2, Clinical Immunology and Allergy\hich\af0\dbch\af37\loch\f0 , Kharkiv, Ukraine}{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid7615703 \hich\af0\dbch\af37\loch\f0 .}{
\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432 
\par }{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid10436759 \hich\af0\dbch\af37\loch\f0 Objective: To study the perfo\hich\af0\dbch\af37\loch\f0 
rmance of intracardiac hemodynamics in patients with arterial hypertension (AH) and chronic obstructive pulmonary disease (COPD).\line \hich\af0\dbch\af37\loch\f0 Material\hich\af0\dbch\af37\loch\f0 s\hich\af0\dbch\af37\loch\f0  an
\hich\af0\dbch\af37\loch\f0 d methods. The study included 21 patients \hich\af0\dbch\af37\loch\f0 ,\hich\af0\dbch\af37\loch\f0  divided into two groups. \hich\af0\dbch\af37\loch\f0 The first\hich\af0\dbch\af37\loch\f0  group included 13 patients with a c
\hich\af0\dbch\af37\loch\f0 ombination of hypertension and COPD (8 males and 5 females, mean age\hich\af0\dbch\af37\loch\f0 \hich\f0  62,03 \'b1\loch\f0  1,85 years), in the second group \hich\af0\dbch\af37\loch\f0 \hich\f0 
 - 10 people (6 men and 4 women, mean age 54,87 \'b1\loch\f0  3 , 05 years) with hypertension without COPD. \hich\af0\dbch\af37\loch\f0 All \hich\af0\dbch\af37\loch\f0 the patient\hich\af0\dbch\af37\loch\f0 s underwent an echo-cardioscopy
\hich\af0\dbch\af37\loch\f0 .
\par \hich\af0\dbch\af37\loch\f0 Re\hich\af0\dbch\af37\loch\f0 sults. The study showed that the value\hich\af0\dbch\af37\loch\f0  of end-diastolic dimension (EDD\hich\af0\dbch\af37\loch\f0 
) of the left ventricle (LV) in patients with hypertension and COPD was \hich\af0\dbch\af37\loch\f0 significantly less \hich\af0\dbch\af37\loch\f0  than in group 2. \hich\af0\dbch\af37\loch\f0 
In patients with a combination of hypertension and COPD compared with those w\hich\af0\dbch\af37\loch\f0 ith hypertension without COPD had significant reductio\hich\af0\dbch\af37\loch\f0 n in end-diastolic volume (EDV) of\hich\af0\dbch\af37\loch\f0 
\hich\f0  LV (98,85 \'b1\loch\f0 \hich\f0  3,21 and 125,50 \'b1\loch\f0  2,1\hich\af0\dbch\af37\loch\f0 2 ml\hich\af0\dbch\af37\loch\f0 , respectively). \hich\af0\dbch\af37\loch\f0 Lower values of minute volumes (MV), cardiac (CV
\hich\af0\dbch\af37\loch\f0 ) volumes and ejection fraction (EF) were determined in patients \hich\af0\dbch\af37\loch\f0 with hypertension and COPD compared with the same parameters in patients with hypertension without COPD. \hich\af0\dbch\af37\loch\f0 
Elevated systoli\hich\af0\dbch\af37\loch\f0 c pulmonary artery pressure (SP\hich\af0\dbch\af37\loch\f0 A\hich\af0\dbch\af37\loch\f0 P\hich\af0\dbch\af37\loch\f0 ) in patients with COPD in the presence of hypertension.
\par \hich\af0\dbch\af37\loch\f0 Conclusions}{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid9450227 \hich\af0\dbch\af37\loch\f0 . }{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid10436759 
\hich\af0\dbch\af37\loch\f0 Thus, more complex hemodynamic conditions for the functioning of the heart in comorbidity lead to much more severe h\hich\af0\dbch\af37\loch\f0 emodynamic disturbances of right ventricular\hich\af0\dbch\af37\loch\f0 
 and the pulmonary circulation and accel\hich\af0\dbch\af37\loch\f0 erate the development of the chronic pulmonary heart\hich\af0\dbch\af37\loch\f0 .}{\f0\fs28\lang1033\langfe1049\langnp1033\langfenp1049\insrsid9133432\charrsid7800674 
\par }\pard \qj \li0\ri0\sa200\sl360\slmult1\widctlpar\aspalpha\aspnum\faauto\adjustright\rin0\lin0\itap0\pararsid7802150 {\fs28\lang1033\langfe1033\langnp1033\insrsid9133432\charrsid7615703 
\par }}